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Study Disproves CDC's Primary Justification for Vaccination by Sayer Ji
According to the Centers for Disease Control and Prevention (CDC), "Immunity to a disease is achieved through the presence of
antibodies to that disease in a person’s system."
[i]
This, in fact, is the main justification for using vaccines to "boost"
immunity, and a primary focus of vaccine research and development.
And yet, newly publish research has revealed that in some cases
no antibodies are required for immunity against some viruses.
Published in the journal
Immunity in March, 2011, and
titled, "B cell maintenance of subcapsular sinus macrophages protects
against a fatal viral infection independent of adaptive immunity,"
researchers found that mice infected with vesicular stomatitis virus
(VSV) can suffer fatal invasion of their central nervous system even in
the presence of high concentrations of "neutralizing" antibodies against
VSV.
[ii]
The researchers found that while B-cells were essential for surviving
a systemic VSV infection through the modulation of innate immunity,
specifically macrophage behavior, the antibodies they produce as part of
the adaptive immune response were "neither needed nor sufficient for
protection." These findings, according to the study authors,
"…contradict the current view that B cell-derived neutralizing
antibodies are absolutely required to survive a primary cytopathic viral
infection, such as that caused by VSV."
The discovery that antibodies are not required for protection against
infection, while counterintuitive, is not novel. In fact, not only are
antibodies not required for immunity, in some cases high levels are
found in the presence of active, even lethal infections. For example,
high serum levels of antibodies against tetanus have been observed
failing to confer protection against the disease. A report from 1992
published in the journal
Neurology found
severe tetanus in immunized patients with high anti-tetanus titers, one of whom died as a result of the infection.
[iii]
These research findings run diametrically opposed to currently held
beliefs regarding the process by which we develop immunity against
infectious challenges. Presently, it is a commonly held view that
during viral infections, innate immunity
must activate
adaptive responses in order to achieve effective immunity. It is
believed that this is why the immune system has developed a series of
innate defenses, including complement, type I interferon, and other
"stopgap measures," which work immediately to lower pathogen burden and
"buy time" for the much slower adaptive immune response to develop.
This view, however, has been called into question by the new study:
"Although this concept may apply to other viral infections, our
findings with VSV turn this view upside down, indicating that during a
primary infection with this cytopathic virus,
innate immunity can be sterilizing without adaptive immune contributions."
Does this strike a mortal blow to the antibody theory which underlies
vaccinology, and constitutes the primary justification for the CDC's
focus on using vaccines to "boost" immunity?
Indeed, in vaccinology, which is the science or method of vaccine
development, vaccine effectiveness is often determined by the ability of
a vaccine to increase antibody titers,
even if this does not translate into real-world effectiveness,
i.e. antibody-antigen matching. In fact, regulatory agencies, such as
the FDA, often approve vaccines based on their ability to raise antibody
titers, also known as "vaccine efficacy," without requiring proof of
vaccine effectiveness, as would seem logical.
The obvious problem with these criteria is that the use of
vaccine adjuvants like mercury,
aluminum hydroxide,
mineral oil, etc. – all of which are intrinsically toxic substances --
will increase antibody titers, without guaranteeing they will
neutralize the targeted antigen, i.e. antibody-antigen affinity. To
the contrary, many of these antibodies lack selectivity, and target
self-structures, resulting in the loss of self-tolerance, i.e.
autoimmunity.
Here is another way of understanding vaccine-induced antibody elevations….
Introducing foreign pathogenic DNA,
chemicals, metals, preservatives, etc., into the body through a syringe
will generate a response not unlike kicking a beehive. The harder you
kick that beehive, the greater will be the "efficacy" (i.e. elevated
antibodies), but the actual affinity that these antibodies will have for
the antigen (i.e. pathogen) of concern is in no way ensured; to the
contrary, the immune response is likely to become misdirected, or
disproportionate to the threat.
Also, valuable immune resources are wasted by generating "false
flag" responses to threats which may not readily exist in the
environment, e.g. there are over 200 forms of influenza A, B & C
which can cause the symptoms associated with annual influenza A,* so the
seasonal trivalent flu vaccine only takes care of little more than 1%
of the possible vectors of infection - and often at the price of
distracting resources away from real threats, as well as exhausting
and/or damaging the entire immune apparatus.
It is clear that one can create a
synthetic immune response
through vaccination, but it is not likely to result in enhanced
immunity, insofar as real-world effectiveness is concerned, which is the
only true judge of whether a vaccine is valuable or not. One might
view the basic criteria used by vaccine researchers, namely, that
generating elevated antibody titers proves the value of the vaccine,
oppositely: proving the vaccine is
causing harm to the body,
especially that of the developing infant and child, by generating
unnecessarily elevated antibodies by any means necessary, i.e. throwing
the chemical and biological kitchen sink at the immune system, e.g.
aluminum, phenol,
diploid (aborted fetal) cells, peanut oil, pertactin, etc.
We leave the reader with a series of quotes addressing the inherent weaknesses of the antibody theory of immunity:
"Just because you give somebody a vaccine, and perhaps get an
antibody reaction, doesn’t mean a thing. The only true antibodies, of
course, are those you get naturally. What we’re doing [when we inject
vaccines] is interfering with a very delicate mechanism that does its
own thing. If nutrition is correct, it does it in the right way. Now if
you insult a person in this way and try to trigger off something that
nature looks after, you’re asking for all sorts of trouble, and we don’t
believe it works."- Glen Dettman Ph.D, interviewed by Jay Patrick, and quoted in "The Great American Deception," Let’s Live, December 1976, p. 57.
"The fallacy of this (antibody theory) was exposed nearly 50 years
ago, which is hardly recent. A report published by the Medical Research
Council entitled 'A study of diphtheria in two areas of Gt. Britain,
Special report series 272, HMSO 1950 demonstrated that many of the
diphtheria patients had high levels of circulating antibodies, whereas
many of the contacts who remained perfectly well had low antibody." -
Magda Taylor, Informed Parent
"Human trials generally correlate "antibody" responses with
protection - that is if the body produces antibodies (proteins) which
bind to vaccine components, then it must be working and safe. Yet Dr
March says antibody response is generally a poor measure of protection
and no indicator at all of safety. "Particularly for viral diseases, the
'cellular' immune response is all important, and antibody levels and protection are totally unconnected."- Private Eye 24/1/2002
"Whenever we read vaccine papers the MD researchers always assume
that if there are high antibody levels after vaccination, then there is
immunity (immunogencity). But are antibody levels and immunity the
same? No! Antibody levels are not the same as IMMUNITY. The recent
MUMPS vaccine fiasco in Switzerland has re-emphasized this point. Three
mumps vaccines-Rubini, Jeryl-Lynn and Urabe (the one withdrawn because
it caused encephalitis) all produced excellent antibody levels but those
vaccinated with the Rubini strain had the same attack rate as those not
vaccinated at all, there were some who said that it actually caused
outbreaks. Ref: Schegal M et al Comparative efficacy of three mumps
vaccines during disease outbreak in Switzerland: cohort study. BMJ,
1999; 319:352-3."- Ted Koren DC
*
PubMed Health

Sayer Ji is the founder of
GreenMedInfo.com. His writings and research have been published in the
Wellbeing Journal, the Journal of Gluten Sensitivity, and have been
featured on numerous websites, including Mercola.com, NaturalNews.com,
Reuters.com, GaryNull.com, and Care2.com.
Disclaimer:
This article is not intended to provide medical advice, diagnosis or
treatment. Views expressed here do not necessarily reflect those of
GreenMedInfo or its staff.